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A Case Study of How Chelation Therapy Cured Hepatitis B

Updated on June 30, 2014

Structure of hepatitis B virion

Having reduced hepatitis B virus to 20,000 copies, chelation therapy enables the immune system to control the virus

One former patient of hepatitis B emailed me recently to share his experience of how he was cured by chelation therapy.

He is from another country who traveled half way around the world because chelation therapy is not available in his country. He was earlier diagnosed as having a chronic hepatitis B. He used the Internet and found my name and my articles on hepatitis B. We had over 40 exchanges of emails before he was convinced to come over to the Philippines for a treatment. He came over in January 2013. He surprised me with an email of his good news last May 2014.

[I will not mention his name because of the mandated confidentiality in treatment for hepatitis B just like for HIV and AIDS. However, being a Chelation Therapy Coach affiliated with Dr. Arturo V. Estuita, MD, a Filipino internist and chelationist, I am entitled to know some of his patients, especially those who contact me to make appointments with Dr. Estuita.]

His first blood test showed that his viral load was over 28,000 international units per milliliter of blood. That is, his viral load was above the threshold of 20,000 international units per ml plus 1 X iLN ALT.

ALT means alanine amino transferase that is an enzyme released by a dead cell killed by the virus. Research in Taiwan for 15 years found that the reliable indicator of hepatitis B that shows a need for treatment is viral load plus ALT (Steven-Huv Han. Hepatitis B: Treatment and Consequences. Internet. June 25,2014). So this is one of the standards in hepatitis B diagnosis and treatment.

His viral load at the tail of his treatment was 22,000. I will not discuss why he wanted to cut short his treatment even at this level. Usually, Dr. Estuita reduces viral load to 10,000 as a double insurance that a patient is cured.

In February 2014 he went for a blood test in his country to monitor his viral load. It was about 17,000. His doctor who had diagnosed him before his chelation was surprised. The doctor required another blood test that showed 8650. (The doctor was not told that his supposed patient had undergone chelation therapy.)

How to explain this cure?

To review: he was a chronic hepatitis B carrier with a viral load of over 28,000. At the tail of his treatment with chelation therapy his viral load was down to 22,000. After one year his viral load is down to 8650. What happened?

One probability is that 22,000 viral load consisted mostly of surface protein and less of DNA infectious particles. A hepatitis B virion consists of surface protein and DNA infectious particle. These can be separated from each other. In an infected person, there are more surface proteins (50 trillion per teaspoon) than DNA infectious particles (500 million per teaspoon), according to Offit and associate (Offit, P. A. MD and L. M. Bell, MD. Vaccines: What Every Parent Should Know. 1999).

When chelation therapy was stopped, the immune system went to work more effectively. The macrophage, a component of immune system, produces an antigen to mark each virion (singular for virus). The virion being an invader stimulates the T-Killer cell to produce an antibody. It uses the antigen as marker to latch itself onto the virion. The antibody changes the size and shape of the virion disabling it to find a hole on cell membrane of the cell of the infected person that matches its size and shape. For this mismatch the virion cannot enter the host cell. So no infection occurs.

The macrophage can engulf a foreign body like a virion, even asbestos. neutrophil is another immunity component that engulfs foreign body. These “eaters” dispose of foreign bodies through the urine. However, these can be overwhelmed by a large population of microbes like the virus that double their population every 20 minutes.

Chelation therapy reduced the population of virus enabling the natural immune system to work Macrophage and neutrophil disposed of more surface proteins from the blood test count of 22,000 to 8650. Below 20,000 viral load, the immune system can control the hepatitis B virus. That is, it can smother the DNA polymerase that is responsible for replication or multiplication of the virion.

In addition the body has developed immunity from the same strain of virus. When hepatitis B virus had been defeated by chelation therapy and the immune system, B cells of this system died off but some remained to become Memory cells. These induce the production of antibodies once they recognize that the same strain of virus is again attacking the body. The body is now immuned. That is how vaccination works.

Offit and Bell recommend that a viral load test must be given about six months after the treatment. One reason is that in three to five months after infection the window period of hepatitis B is still in place. During the window period, blood test cannot catch an antigen. That is a false negative. Without treatment, the virus replicates aggressively that a lot of antigens can be caught in six months, past the window period. This is already the start of chronic stage of hepatitis B when antigens (indicated by HBsAg) show up; and antibodies against the core protein (indicated by IgG-anti-HBc) show up.

Conventional drugs

Conventional drugs like lamivudine, fevoir or interferon cannot completely control hepatitis B virus. Virus develop resistance against these drugs, according to Steven-Huv Han. Drugs target the DNA polymerase that is well protected by the core protein and plasmid.

Chelation therapy

This works by the physical mode. The chelation solution has a negative charge. The virion has a positive charge. The two charges attract and attach to each other resulting in the disposal of the virion through the urine. The virion cannot develop resistance to the charge of the chelation therapy solution.

Be confident

Don’t panic if a blood test still shows positive for antigen (indicated by HBsAg) at count 10,000. The reason is that it is most probable that the antigens are marking empty surface proteins. DNA infectious particles no longer exist. Remember the mathematical extrapolation of 100,000 surface protein is to 1 DNA infectious particle? When the population of hepatitis B virus no longer increases that means that the mathematical extrapolation has become actual. Cure by chelation therapy has materialized.

Hubs related to hepatitis B written by conradofontanilla


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